Chlorisondamine is a nicotinic acetylcholine receptor antagonist that produces both neuronal and ganglionic blockade.

Chlorisondamine has been shown to form noncovalent complexes with various biomolecules including sphingomyelin and other associated glycolipids.[1][2][3]

References

  1. ^ Woods AS, Moyer SC, Wang HY, Wise RA (2003). "Interaction of chlorisondamine with the neuronal nicotinic acetylcholine receptor". Journal of Proteome Research. 2 (2): 207–12. doi:10.1021/pr025578h. PMID 12716135.
  2. ^ Jackson SN, Wang HY, Woods AS, Ugarov M, Egan T, Schultz JA (February 2005). "Direct tissue analysis of phospholipids in rat brain using MALDI-TOFMS and MALDI-ion mobility-TOFMS". Journal of the American Society for Mass Spectrometry. 16 (2): 133–8. Bibcode:2005JASMS..16..133J. doi:10.1016/j.jasms.2004.10.002. PMID 15694763. S2CID 5894935.
  3. ^ Woods AS, Ugarov M, Egan T, Koomen J, Gillig KJ, Fuhrer K, Gonin M, Schultz JA (April 2004). "Lipid/peptide/nucleotide separation with MALDI-ion mobility-TOF MS". Analytical Chemistry. 76 (8): 2187–95. doi:10.1021/ac035376k. PMID 15080727.
Allikas: https://en.wikipedia.org/wiki/Chlorisondamine
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